This webinar discusses how autophagy-dependent metabolic reprogramming may rescue the antiviral potency of anti-HIV-1 T-cells in subjects under antiretroviral therapy (ART). Although conventional ART manages to suppress HIV-1 infection’s progression, it does not restore a proper T-cell’s immunity. Here, we propose that autophagy is a pivotal key process to mount an effective T-cell responses and metabolic fitness by providing diverse carbon sources within a hostile environment during persistent HIV-1 infection.
 

Tune in to learn more about		How autophagy confers differential metabolic reprogramming in a T-cell subset-dependent manner. How autophagy is regulated through IL-21 and AMPK signaling in T-cells, providing the proper mitochondrial bioenergetics requirement. Autophagy as a valuable therapeutic tool to enhance T-cells antiviral immunity.
DATE		Thursday, September 16, 2021
TIME		08:00 PT (Los Angeles) 11:00 ET (New York) 15:00 GMT (London)
		Register for the webinar now
		For Research Use Only. Not for use in diagnostic procedures.

	PRESENTED BY
		Hamza Loucif PhD Candidate INRS-Centre Armand-Frappier Santé Biotechnologie, Laval, Quebec, Canada Agilent Technologies, Inc